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In addition, ferroptosis is manifested by a decrease in the activity of the regulatory core enzyme GPX4 of the glutathione antioxidant system (54)

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Simultaneously, BCAA-mediated mTORC1 hyperactivation in podocytes suppresses autophagy and exacerbates insulin resistance, creating a metabolic milieu that potentiates toxin-induced injury (136, 149)

Together, they can help keep cells functioning properly and be more resilient over time