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Within gene regulatory regions, the acetylation of lysine residues of histone tails has been correlated with activation of transcription

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Both SP and L-car attenuated DOX-induced ovarian oxidative damage, subsequent ovarian mtDNA expression, and activation of downstream inflammatory and apoptotic pathways, particularly lowering P53 and caspase-3 protein expressions in preantral follicles, thus preventing ovarian insult and follicle demise and extending the post-chemotherapy fertile period