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Given the NCOA4 abundance is dually modulated by autophagy and the ubiquitin-proteasome system, its downregulation might indicate a feedback mechanism involving ubiquitin-dependent proteasomal degradation, which is promoted by high iron levels and involves iron-dependent binding to the E3 ubiquitin ligase HERC2 (137)

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Role of growth hormone, insulin-like growth factor 1 and insulin-like growth factor-binding protein 3 in development of non-alcoholic fatty liver disease