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Notably, low concentrations of O can also regulate mitochondrial function by activating PGC-1, thereby enhancing fatty acid oxidation [191, 192]

The most actionable approach is tracking the downstream markers that respond to glutathione status: oxidative stress markers, liver enzymes, methylation markers, and inflammation

After confirming that the 5% water content suppository exhibited optimal delivery, further fluorescence staining and observation of rectal tissue following administration of the 5% water content suppository were performed

[168], he proposed that NO may bind to the enzymes of mitochondrial ETC, such as NADH succinate oxidoreductase, NADH ubiquinone oxidoreductase, and cis aconitase and induce uncoupling of OXPHOS followed by the inhibition of glycolysis [66] and depletion of ATP in the cells [169]